Promote Scientific Collaboration
Access to Unique Applications and Tools
HDinHD: Open Source Science for the HD Research Community
The goal of HDinHD is to foster and support a collaborative community united in its drive to accelerate the development of therapeutics that will delay the onset and/or ameliorate the effects of Huntington’s disease. HDinHD seeks to achieve this through:
- Sharing HD-related primary scientific data
- Sharing analyses and computational models built from HD-related scientific data
- Providing browsing and data interrogation tools over both primary and analyzed data that facilitate data exploration and hypothesis generation
- Building a forum for HD researchers to highlight their data, tools, know-how and insight to the community
The HDinHD website is currently a joint development effort by CHDI and UCLA, through the laboratory of Dr. Giovanni Coppola. As you will see when you Register for access, HDinHD highlights data and tools provided by the broader community. If you are interested in Contributing Data to HDinHD, suggest links to other HD-related scientific data or websites, or would otherwise like to provide Feedback to us, we encourage you to do so. HDinHD is for the community; please help us tailor and grow HDinHD in a direction that can make a difference to your research, and ultimately, to improve the lives of HD families.
Latest news and publications from around the HD Ecosystem
- Has a “wonder drug” for dementia been discovered? (Spoiler alert: no.)
- Huntington’s Disease Therapeutics Conference 2017 – Day 3
- Huntington’s Disease Therapeutics Conference 2017 – Day 2
- Huntington’s Disease Therapeutics Conference 2017 – Day 1
- Enemy at the gates – huntingtin disrupts nuclear transport
- Fibril polymorphism affects immobilized non-amyloid flanking domains of huntingtin exon1 rather than its polyglutamine core.
- Functionalisation of Polyvinylpyrrolidone on Gold Nanoparticles Enhances Its Anti-Amyloidogenic Propensity towards Hen Egg White Lysozyme.
- 7,8 Dihydroxyflavone ameliorates cognitive and motor deficits in a Huntington’s disease mouse model through specific activation of the PLCγ1 pathway.
- The human body as an energetic hybrid? New perspectives for chronic disease treatment?
- Disease-related gene module detection based on a multi-label propagation clustering algorithm.
- Professor Serbin Invited to Vatican to Meet Pope Francis
- Taube Philanthropies Donates $3 Million to Stanford, Gladstone for Huntington’s Research
- Northumberland resident says physician-assisted dying laws need to be all encompassing
- EXCLUSIVE: ‘We never expected we would grow old together’: Seven Year Switch’s Stacey Louise …
- Voyager Therapeutics Reports First Quarter 2017 Financial Results and Corporate Highlights