Promote Scientific Collaboration
Access to Unique Applications and Tools
HDinHD: Open Source Science for the HD Research Community
The goal of HDinHD is to foster and support a collaborative community united in its drive to accelerate the development of therapeutics that will delay the onset and/or ameliorate the effects of Huntington’s disease. HDinHD seeks to achieve this through:
- Sharing HD-related primary scientific data
- Sharing analyses and computational models built from HD-related scientific data
- Providing browsing and data interrogation tools over both primary and analyzed data that facilitate data exploration and hypothesis generation
- Building a forum for HD researchers to highlight their data, tools, know-how and insight to the community
The HDinHD website is currently a joint development effort by CHDI and UCLA, through the laboratory of Dr. Giovanni Coppola. As you will see when you Register for access, HDinHD highlights data and tools provided by the broader community. If you are interested in Contributing Data to HDinHD, suggest links to other HD-related scientific data or websites, or would otherwise like to provide Feedback to us, we encourage you to do so. HDinHD is for the community; please help us tailor and grow HDinHD in a direction that can make a difference to your research, and ultimately, to improve the lives of HD families.
Latest news and publications from around the HD Ecosystem
- HD and Histamines: Targeting Hybrid Receptors to Quiet Stressful Brain Talk
- Changing jobs: converting other cell types into neurons
- HD Young Adult Study defines the sweet spot: symptom-free with measurable changes
- Fountain of youth: HTT protein repairs neurons by maintaining youthful state
- What does COVID-19 mean for Huntington’s disease families and HD research?
- TBK1 phosphorylates mutant Huntingtin and suppresses its aggregation and toxicity in Huntington's disease models.
- Decreased Myocyte Enhancer Factor 2 Levels in the Hippocampus of Huntington's Disease Mice Are Related to Cognitive Dysfunction.
- Impaired intracellular trafficking of sodium-dependent vitamin C transporter 2 contributes to the redox imbalance in Huntington's disease.
- Editorial: Role of Protein Palmitoylation in Synaptic Plasticity and Neuronal Differentiation.
- DNA Methyltransferase 1 (DNMT1) Acts on Neurodegeneration by Modulating Proteostasis-Relevant Intracellular Processes.
- I've Learned to Focus on Living in the Moment
- COVID-19 impact on CNS Specific Antisense Oligonucleotide Market analysis, industry trends ...
- Squid Gene-Editing Shows New Possibilities For Treating Genetic Diseases
- Huntington's Disease Society of America's Wisconsin Chapter to host Madison Team Hope Walk
- Support for Astros pitcher Joe Smith after his mom's passing