Promote Scientific Collaboration
Access to Unique Applications and Tools
HDinHD: Open Source Science for the HD Research Community
The goal of HDinHD is to foster and support a collaborative community united in its drive to accelerate the development of therapeutics that will delay the onset and/or ameliorate the effects of Huntington’s disease. HDinHD seeks to achieve this through:
- Sharing HD-related primary scientific data
- Sharing analyses and computational models built from HD-related scientific data
- Providing browsing and data interrogation tools over both primary and analyzed data that facilitate data exploration and hypothesis generation
- Building a forum for HD researchers to highlight their data, tools, know-how and insight to the community
The HDinHD website is currently a joint development effort by CHDI and UCLA, through the laboratory of Dr. Giovanni Coppola. As you will see when you Register for access, HDinHD highlights data and tools provided by the broader community. If you are interested in Contributing Data to HDinHD, suggest links to other HD-related scientific data or websites, or would otherwise like to provide Feedback to us, we encourage you to do so. HDinHD is for the community; please help us tailor and grow HDinHD in a direction that can make a difference to your research, and ultimately, to improve the lives of HD families.
Latest news and publications from around the HD Ecosystem
- Ask the expert: Q&A on the huntingtin-lowering trial program
- Success! ASO drug reduces levels of mutant protein in Huntington’s disease patients
- Switch off the genome editor when you’re done
- A new way of thinking about trials to prevent Huntington’s disease
- New roles for huntingtin: removing a healthy protein to understand its function
- Nanostructural Differentiation and Toxicity of Amyloid-β25-35 Aggregates Ensue from Distinct Secondary Conformation.
- Generation of induced pluripotent stem cell line, CSSi002-A (2851), from a patient with juvenile Huntington Disease.
- 7,8-dihydroxyflavone ameliorates cognitive and motor deficits in a Huntington’s disease mouse model through specific activation of the PLCγ1 pathway.
- Pearls & Oy-sters: Family history of Huntington disease disguised a case of dentatorubral-pallidoluysian atrophy.
- Regulation of HSF1 protein stabilization: an updated review.
- Dean Investment Associates Lifted By $508860 Its Price T Rowe Group (TROW) Holding; 10 …
- Head-To-Head Analysis: GTX (GTXI) & Wave Life Sciences (NASDAQ:WVE)
- uniQure Advancing AMT-130 Development in the New Year
- uniQure Aims to Begin First Human Trial of Investigational Gene Therapy AMT-130 for Huntington’s …
- H. Lundbeck A/S- Expected to Post FY2017 Earnings of $2.44 Per Share (HLUYY)